By Aayushi Gupta
Published Feb 3, 2024
HPV, particularly high-risk strains like HPV 16 and 18, is the leading cause of cervical cancer. HPV is a sexually transmitted infection, and persistent infection with high-risk types increases the risk of cervical cancer.
Smokers are at a higher risk of developing cervical cancer compared to non-smokers. Tobacco smoke contains carcinogens that can damage cervical cells, increasing the risk of cancer development.
Individuals with weakened immune system, such as those with HIV/AIDS, are at an increased risk of cervical cancer. A compromised immune system is less effective at fighting HPV infections.
Engaging in sexual activity at an early age increases the likelihood of HPV infection, which is a significant risk factor for cervical cancer.
Having multiple sexual partners increases the risk of HPV exposure, which in turn raises the risk of cervical cancer. The more sexual partners you have, the higher your risk of acquiring HPV infections.
Women with a family history of cervical cancer may have a higher risk of developing the disease themselves. However, not all types of cervical cancer are hereditary.
Chlamydia infection can cause chronic inflammation and cellular damage in the cervix, which may contribute to the development of cervical cancer over time.
Prolonged use of oral contraceptives (birth control pills) has been associated with an increased risk of cervical cancer. However, this risk typically decreases once a woman stops taking the pill.
Not undergoing regular Pap smear tests or HPV screenings reduces the chances of early-stage cervical cancer, increasing the risk of progression to advanced stages.